Sometimes, it really depends on if there is cardiac damage done, angina is cause by less oxygenated blood reaching the arteries that circle the outside of the heart. This in turn cause pain, when you take nitroglycerin, this causes your veins to vasodialate or in a sense get bigger for a short period of time, this increases the blood flow, and cause the angina to decrease, if the angina is cause by a blockage, then triponin, ckmb, and other cardiac enzymes will rise, but ckmb will rise with any muscle damage, the best indicator of ischemia or heart muscle damage is triponin, where i work if it goes over 0.03 then it it indicative of a mild to moderate heart attack
Not unless you have a heart attack and have damage to the heart muscle. When the heart muscle loses oxygenated blood, like when you have a blocked coronary artery, the area where it is not getting blood begins to die, that is when your cardiac enzyme levels will rise. that is how they determine that you have had a heart attack. By the time the levels rise, the damage is already done.
Angina is manageable most of the time with medication, diet and exercise. however if you have angina, and you can not get the chest pain to go away with your meds, or rest, then you need to get to the ER immediately. Especially if the pain radiates in to your left arm or up in to your jaw. If you get quick enough treatment, they can stop a heart attack in it's tracks, before too much damage is done to the heart muscle.
To bubli: If there is ST depression most definately an MI has occured. have you done cardic cath to determine if pt. has any occluded arteries? If not, pt needs referral to Cardiologist for cardiac cath/poss PTCA.
enzymatic test of SGOT/AST is a definite test for MI and Angina…. CPK-MB is also advisable testing because it will rise 1-3 hours after myocardial injury.
SLDH (particularyl 1 and 2) may be used as late diagnosis of MI, it would rise 2-3 days after myocardial injury
Troponin 1 will also be the best diagnostic exam!
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